Of myocytes So how do you control your movement?

肌细胞 那么你如何控制你的运动呢？

The effect this has on excitability of myocytes, however, is complex.

然而，这对心肌细胞兴奋性的影响是复杂的。

Markers are substances released into the bloodstream upon death of cardiac myocytes.

标记物是心肌细胞死亡后释放到血液中的物质。

While initial changes seem to increase myocyte excitability; further rise of potassium has the OPPOSITE effect.

初始变化可能增加心肌细胞的兴奋性，但钾进一步升高却具有相反的作用。

They have the ability to differentiate into myocytes with structural and functional properties of cardiomyocytes and into many other cell types.

它们具有分化成具有心肌细胞的结构和功能特性的肌细胞以及许多其他细胞类型的能力。

These drugs bind to and block the fast sodium channels that are responsible for the depolarizing phase and contractile myocytes.

这些药结合并阻断快钠通道。快钠通道作用于去极化时相和有收缩性的心肌。

And this is due to mutations in the TSC1 or TSC2 tumor suppressor genes, genes that usually suppress tumor growth, therefore a mutation leads to myocyte overgrowth.

由于TSC1和TSC2抑癌基因的突变，这些基因通常抑制肿瘤生长，因此此类基因突变可导致肌细胞过度生长。

However because calcium is also involved in cardiac myocyte contraction, these agents also reduce contractility of the heart and should not be used in case of systolic heart failures.

然而，因为钙通道也参与了心肌收缩，所以这些药物也会减弱心脏的收缩力，因此收缩性心力衰竭患者不应该使用该类药物。