But, as I mentioned earlier, this renin angiotensin aldosterone system is stimulated by low salt intake.

但是，正如我之前提到的，低盐饮食会导致 RAA 系统活跃。

Using its protein spike, it latches onto a cell-surface receptor called Angiotensin-Converting Enzyme 2, or ACE2.

它利用其蛋白质刺突锁定在称为血管紧张素转换酶 2 或 ACE2 的细胞表面受体上。

This system begins with your liver releasing angiotensinogen, and, after a series of steps that you can read about in an endocrinology textbook, angiotensin I is produced.

该系统开始从肝脏释放血管紧张素原，经过一系列步骤（请参考内分泌相关教材）之后，I 型血管紧张素产生了。

Your kidneys cook up hormones like renin and angiotensin which help regulate levels of sodium and fluids in your body, and also help expand and constrict blood vessels.

你的肾脏会分泌肾素和血管紧张素等荷尔蒙，这些荷尔蒙有助于调节体内钠和液体的水平，也有助于扩张和收缩血管。

Along the same lines, drugs that reduce the effect of aldosterone can also cause hyperkalemia, and these are drugs like renin inhibitors, ACE inhibitors, angiotensin II receptor antagonists, selective aldosterone blockers, and potassium-sparing diuretics.

同理，可以降低醛固酮效能的药物也可以导致高钾血症，例如肾素抑制剂、ACE抑制剂、血管紧张素II受体拮抗剂、选择性醛固酮受体阻滞剂以及保钾利尿剂等。